Department of Pathology & Laboratory Medicine
David Witte, M.D.
Acting Chairman
MD - University of Cincinnati, 1979; Specialty Training - Anatomic pathology, Washington University, St. Louis and Pediatric Pathology, Children's Hospital Medical Center, Cincinnati.Research Interests - Gene expression and regulation during development, molecular pathology; Specialty Area - Pediatric pathology.
Recent Publications:
Welch TR, Frenzke M, and Witte DP. Evidence of a role for local complement expression in a model of progressive glomerulonephritis. Pediatric Research 48: 200-5, 2000.West CD, McAdams AJ, Witte DP. Acute non-proliferative glomerulitis: A cause of renal failure unique to children. Pediatric Nephrology 14: 786-793, 2000.
Steinbrecher KA, Tuohy TM, Heppner Goss K, Scott MC, Witte DP, Groden J, Cohen MB. Expression of Guanylin Is Downregulated in Mouse and Human Intestinal Adenomas.Biochem Biophys Res Commun. 2000 Jun 24;273 (1):225-230.
McGraw D, Forbes S, Mak J, Witte D, Carrigan P, Leikauf G, and Liggett S. Airway contractility is decreased by transgenic overexpression of the (2-Adrenergic receptor in airway epithelial cells. Am J. Physiol Lung Cell Mol Physiol…279:L379-L389, 2000.
McLaughlin L, Mistry M, Zhu G, Ley-Ebert C, Stuart WD, Florio C, Groen P, Witt SA, Kimbal TR, Witte DP, Harmony JAK, Aronow BJ. ApolipoproteinJ/clusterin limits the severity of murine autoimmune myocarditis. J Clin Invest. 106:1105-1113, 2000.
West C, Witte D, McAdams A. Composition of the nephritic factor generated glomerular deposits in membranoproliferative glomerulonephritis type II. Am J Kid Dis (in press).
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